anti ca1 (Proteintech)
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Proteintech
anti ca1
Anti Ca1, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 14 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+ca1/CA1+Antibody/pm41673867-132-13-15
Average 93 stars, based on 14 article reviews
Anti Ca1, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 14 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+ca1/CA1+Antibody/pm41673867-132-13-15
Average 93 stars, based on 14 article reviews
anti ca1 - by Bioz Stars,
2026-09
93/100 stars
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Membrane:Article Title: Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway. Article Snippet: .. After sealing, the membrane was incubated overnight with primary antibodies at 4 °C: Article Title: Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway Article Snippet: .. After sealing, the membrane was incubated overnight with primary antibodies at 4 °C: Incubation:Article Title: Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway. Article Snippet: .. After sealing, the membrane was incubated overnight with primary antibodies at 4 °C: Article Title: Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway Article Snippet: .. After sealing, the membrane was incubated overnight with primary antibodies at 4 °C: Phospho-proteomics:Article Title: Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway. Article Snippet: .. After sealing, the membrane was incubated overnight with primary antibodies at 4 °C: Article Title: Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway Article Snippet: .. After sealing, the membrane was incubated overnight with primary antibodies at 4 °C: other:Article Title: Quantitative Proteomic Analysis of Mouse Sciatic Nerve Reveals Post-injury Upregulation of ADP-Dependent Glucokinase Promoting Macrophage Phagocytosis Article Snippet: The following primary antibodies were used in this study: anti-ADPGK (15639-1-AP), anti-RAC2 (10735-1-AP), anti-CA1 (13198-2-AP) and anti-MPO (22225-1-AP) from Article Title: M1-Type Macrophages Secrete TNF-α to Stimulate Vascular Calcification by Upregulating CA1 and CA2 Expression in VSMCs Article Snippet: The proteins were transferred to a PVDF membrane (Millipore, Burlington, MA, USA), which was then blocked with 5% nonfat milk for 2 h. The membrane was incubated overnight at 4°C with primary antibodies against CA1, Article Title: M1-Type Macrophages Secrete TNF-α to Stimulate Vascular Calcification by Upregulating CA1 and CA2 Expression in VSMCs Article Snippet: The proteins were transferred to a PVDF membrane (Millipore, Burlington, MA, USA), which was then blocked with 5% nonfat milk for 2 h. The membrane was incubated overnight at 4°C with primary antibodies against CA1, |
![Establishment of <t>CA1</t> KI ApoE [−/−] C57BL/6 mice via the CRISPR/Cas9 gene re-editing technique. A. Diagram of the establishment of KI mice with CA1 cDNA inserts. An expression cassette with a CAG promoter and CA1-encoding gene was inserted into intron 1 of the Rosa26 locus in the C57BL/6 mouse chromosome via the CRISPR/Cas9 system. B. Representative PCR images showing the successful establishment of CA1-overexpressing KI mice. a. PCR with primer set 1 yielded one band of 296 bp in KI mice. b. PCR with primer set 2 yielded one band of 335 bp in KI mice. c. PCR with primer set 3 yielded one band of 245 bp in the KI mice. KI: knock-in mice; WT: wild type; M: molecular size marker of DNA.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_9080/pmc11999080/pmc11999080__gr1.jpg)
![Changes in the body weights of the AS model mice. CA1-overexpressing ApoE [−/−] mice and ordinary ApoE [−/−] mice were induced to AS with high-fat food. These patients were divided into the following groups: normal, AS model, AS model with MTZ treatment and AS model with MTZ preventive-treatment. Each group included 15 mice. Mice with CA1 overexpression generally had greater body weights than did those without CA1 overexpression. The weight of the mice with AS was generally greater than that of the mice without AS, and the weight of the mice with AS was reduced following MTZ treatment and MTZ-preventive treatment.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_9080/pmc11999080/pmc11999080__gr2.jpg)
![Biochemical examination of mouse peripheral blood. Compared with those in healthy controls, HDL levels were significantly lower, and AI, LDL, TC and TG levels were elevated in AS mice. The levels of these indices were restored after MTZ treatment. Moreover, the HDL level was significantly lower and the levels of AI, LDL, TC and TG were greater in CA1-overexpressing mice than in ApoE [−/−] mice with normal CA1 expression, regardless of whether these mice were induced to develop AS or treated with MTZ. ∗ indicates P < 0.05, ∗∗ indicates P < 0.01, ∗∗∗ indicates P < 0.001 and ∗∗∗∗ indicates P < 0.0001.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_9080/pmc11999080/pmc11999080__gr6.jpg)
![Immunostaining of CA1 expression in mouse aortic tissues. Immunohistochemistry revealed CA1 expression (brown) in the aortic plaques of the animals with AS (↓). CA1 was also weakly expressed in aortic VSMCs (↓↓). Immunofluorescent immunohistochemistry revealed that CA1 levels were increased in the cardiac aorta tissues of CA1-overexpressing ApoE [−/−] mice, regardless of whether the KI mice had induced AS, compared with those of ordinary ApoE [−/−] mice. The quantified signal is normalized to the total cellularized area. ∗∗∗ indicates P < 0.001 and ∗∗∗∗ indicates P < 0.0001.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_9080/pmc11999080/pmc11999080__gr7.jpg)
![Calcification of mouse cardiac aorta tissues via Von Kossa staining. Extensive calcium deposition was detected in cardiac aorta tissues from CA1-overexpressing AS and AS mice following MTZ treatment. Little calcification was observed in the aortic tissues of ordinary ApoE [−/−] mice with induced AS.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_9080/pmc11999080/pmc11999080__gr8.jpg)
![Immunofluorescence analysis of macrophages in mouse cardiac aorta tissues. The aortic tissues were stained with DAPI (blue). CD86 (green) represents the M1 macrophage subtype, and CD163 (yellow) represents the M2 macrophage subtype. The expression levels of CD86 were semiquantitatively analyzed. Higher CD86 expression was detected in the aortic tissues of CA1-overexpressing mice than in those of ordinary Apoe [−/−] mice. Increased CD86 expression was detected in the aortic tissues of both CA1-overexpressing mice and ordinary ApoE [−/−] mice when they were induced to AS. ∗ indicates P < 0.05, ∗∗ indicates P < 0.01, ∗∗∗ indicates P < 0.001 and ∗∗∗∗ indicates P < 0.0001.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_9080/pmc11999080/pmc11999080__gr9.jpg)
